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GCC Nursing Guide — Eating Disorders
Psychiatry & Medicine GCC Context DSM-5 / MARSIPAN / NICE Updated Apr 2026
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DSM-5 Eating Disorder Classification

Anorexia Nervosa (AN)Restriction, distorted body image
Bulimia Nervosa (BN)Binge-purge cycles, normal/high weight
Binge Eating Disorder (BED)Binges without compensatory purging
ARFIDAvoidant/Restrictive Food Intake Disorder
OSFEDOther Specified Feeding/Eating Disorder

Anorexia Nervosa — Subtypes

Restrictive (AN-R)

Weight loss exclusively by dieting, fasting, excessive exercise. No binge-purge behaviour. Most medically severe at low weights.

Binge-Purge (AN-BP)

Meets AN criteria (low weight, fear of gain) but also engages in binge/purge episodes. Electrolyte abnormalities more prominent.

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AN Diagnostic Criteria (DSM-5)

  1. Energy restriction — significantly low body weight relative to age, sex, developmental trajectory, and physical health. BMI <17.5 kg/m² is the ICD-10/clinical threshold; DSM-5 does not specify a BMI cutoff.
  2. Intense fear of weight gain — persistent fear of becoming fat or weight gain behaviour even when underweight.
  3. Distorted body image — disturbed experience of body weight/shape; undue influence of weight/shape on self-evaluation; persistent lack of recognition of severity of low weight.

AN Severity (DSM-5 BMI-based)

MildBMI ≥17 kg/m²
ModerateBMI 16–16.99
SevereBMI 15–15.99
ExtremeBMI <15 kg/m²
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Physical Signs — Anorexia Nervosa

Integumentary
  • Lanugo hair — fine downy hair on trunk/limbs (thermoregulation response)
  • Dry skin, hair loss (telogen effluvium), brittle nails, yellow skin (hypercarotinaemia)
  • Russell's sign — NOT in AN-R; seen in purging (callosities on dorsum of hand)
Cardiovascular & Autonomic
  • Bradycardia — resting HR <60; HR <50 = medical admission threshold
  • Hypotension — systolic <90 mmHg; postural drop >10 mmHg
  • Hypothermia — core temp <35.5°C
  • Peripheral oedema (refeeding or protein deficiency)
Reproductive & Endocrine
  • Amenorrhoea — hypothalamic suppression; may persist post-weight restoration
  • Low LH, FSH, oestrogen
  • Low T3 (sick euthyroid syndrome)
  • Low IGF-1, elevated cortisol
  • Delayed puberty in adolescents
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Physical Signs — Bulimia Nervosa

Oral & Salivary
  • Dental erosion — perimolysis (acid erosion of lingual surfaces of upper teeth from self-induced vomiting)
  • Parotid gland enlargement — bilateral, painless swelling; elevated amylase
  • Sialadenosis — submandibular swelling
Hands & Electrolytes
  • Russell's sign — calluses/scarring on dorsal knuckles from repeated self-induced vomiting
  • Hypokalaemia — most significant electrolyte abnormality; causes arrhythmias, muscle weakness
  • Hypochloraemia, metabolic alkalosis
  • Hypomagnesaemia, hyponatraemia
ARFID — Key Features

Avoidance of food based on sensory characteristics (texture, colour, smell), fear of choking/vomiting, or apparent lack of interest. NOT driven by body image concerns. Common in autism, anxiety disorders. Significant nutritional deficits without deliberate weight loss intent.

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MARSIPAN — Medical Risk Stratification

MARSIPAN (Management of Really Sick Patients with Anorexia Nervosa) provides a structured framework for assessing medical risk and determining appropriate care setting.

Parameter Low Risk Medium Risk High Risk — Admit
BMI (adults)≥1513–14.9<13
Heart rate (resting)≥6050–59<50
Systolic BP≥10090–99<90
Postural drop<5 mmHg5–10 mmHg>10 mmHg
Temperature≥35.5°C34.5–35.4°C<34.5°C
Muscle functionSit up unaidedDifficulty risingCannot rise from squat
QTc interval<450 ms450–470 ms>470 ms
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Any single HIGH RISK criterion = medical admission required. Do not manage in community. Involve physician and eating disorder team immediately.

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GCC Context — Cultural Considerations

Prevalence & Stigma

Eating disorders are significantly underreported across GCC countries due to cultural stigma, family shame, and low awareness among primary care providers. Families often perceive low weight as a spiritual or physical illness rather than psychiatric.

Increasing prevalence has been documented among Gulf women, particularly university-aged students in Saudi Arabia, UAE, and Kuwait. Rates comparable to Western populations in younger cohorts.

Social Media & Body Image

Rapid adoption of social media platforms — Instagram, TikTok, Snapchat — is directly linked to increasing body dissatisfaction among Gulf adolescents. "Fitspiration" culture, filters, and influencer culture drive unrealistic body ideals.

Social media pressure Western beauty ideals Family weight comments Competitive thinness Weight stigma
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Anorexia Nervosa has the highest mortality rate of any psychiatric disorder — approximately 10% (5–6% from medical complications, 2–5% suicide). Early identification and medical monitoring are critical.

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Cardiovascular Complications

  • Resting HR <50 bpm = medical admission threshold (MARSIPAN)
  • Sinus bradycardia most common; sick sinus syndrome possible
  • QTc prolongation — electrolyte-mediated (especially hypokalaemia, hypomagnesaemia); fatal arrhythmia risk. QTc >470 ms = high risk
  • Ventricular tachycardia risk during refeeding; monitor ECG
  • Cardiac muscle mass reduced proportionally to body weight loss
  • Decreased left ventricular mass and chamber size on echocardiogram
  • Reduced cardiac output — contributes to hypotension and exercise intolerance
  • Mitral valve prolapse reported — may regress with weight restoration
  • Pericardial effusion in severe cases
  • Systolic BP <90 mmHg at rest — admission criterion
  • Postural hypotension — systolic drop >10 mmHg on standing
  • Syncope risk — advise slow position changes; fall prevention
  • Autonomic dysfunction contributes to HR and BP dysregulation

Electrolyte Abnormalities

Electrolyte AN BN / AN-BP Clinical Risk
Potassium (K+)Low-normal or lowSignificantly low (purging)Arrhythmias, muscle weakness, ileus
PhosphateLow (especially refeeding)Low-normalRefeeding syndrome, cardiac failure
MagnesiumLowLow (laxative misuse)Arrhythmias, seizures, worsens hypokalaemia
SodiumLow (water loading)Low or normalHyponatraemic encephalopathy
ChlorideNormalLow (vomiting)Metabolic alkalosis
CalciumLow (poor intake)NormalOsteoporosis, tetany
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Bone Complications

Osteoporosis is one of the most significant long-term complications of AN, driven by prolonged oestrogen deficiency, low calcium/vitamin D intake, elevated cortisol, and IGF-1 deficiency.

  • Bone loss is rapid — detectable within 6–12 months of amenorrhoea
  • Peak bone mass is not achieved in adolescents with AN — lifelong consequence
  • DEXA scan recommended after 1 year of amenorrhoea or significant weight loss
  • Vitamin D supplementation 800–2000 IU/day; calcium 1000–1200 mg/day
  • Bisphosphonates generally avoided in premenopausal women
  • Weight restoration and oestrogen recovery is the primary treatment
  • Stress fractures risk — limit high-impact exercise
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Haematological & Renal

Haematological

  • Pancytopenia — bone marrow suppression/hypoplasia in severe, prolonged AN; gelatinous bone marrow transformation
  • Anaemia, leukopenia, thrombocytopenia
  • Impaired immune function — increased infection susceptibility

Renal

  • Hypokalaemic nephropathy — chronic hypokalaemia from purging causes vacuolar tubular changes and interstitial fibrosis
  • Pre-renal AKI from dehydration — especially with laxative/diuretic misuse
  • Renal calculi — calcium oxalate (low urine volume)
  • Hyposthenuria (inability to concentrate urine) in chronic cases
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Neurological Complications

Structural
  • Brain atrophy — cortical grey and white matter volume loss in severe AN; partially reversible with weight restoration
  • Enlarged CSF spaces, sulcal widening on MRI
  • Structural changes most prominent in chronic cases
Cognitive
  • Impaired executive function, concentration, working memory
  • Rigid thinking patterns — cognitive inflexibility perpetuates illness
  • Impaired social cognition and emotional recognition
  • Improved but not always fully normalised with recovery
Peripheral
  • Peripheral neuropathy — thiamine deficiency (Wernicke's risk)
  • Myopathy — muscle wasting, proximal weakness
  • Autonomic neuropathy — HR and BP dysregulation
  • Seizures — hypoglycaemia, hyponatraemia, or refeeding
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Monitoring Schedule

Inpatient — Daily
  • Electrolytes: U&E, phosphate, magnesium, calcium (especially first 2 weeks of refeeding)
  • Blood glucose (hypoglycaemia risk)
  • ECG if any electrolyte abnormality or QTc prolongation detected
  • Fluid balance, urine output
  • Vital signs (HR, BP including postural, temperature) every shift
Weight Monitoring
  • Weigh 3 times per week — same time, same clothes, post-void
  • Expect weight fluctuation from fluid shifts — interpret with caution
  • Target: 0.5–1 kg/week weight gain inpatient
Ongoing / Outpatient
  • ECG: at baseline; repeat if electrolytes abnormal or medications added (especially antipsychotics, SSRIs)
  • DEXA scan: after 1 year amenorrhoea or significant weight loss
  • Bone profile, vitamin D, ferritin, B12, folate: quarterly
  • Thyroid function (low T3 sick euthyroid syndrome)
  • FBC: monitor for pancytopenia
  • BMI: at every clinical contact
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Refeeding syndrome is potentially fatal. It occurs when carbohydrate-driven insulin surge causes massive intracellular shift of phosphate, potassium, and magnesium, leading to hypophosphataemia, fluid shifts, and multi-organ failure. Prevention requires slow caloric escalation and proactive electrolyte/vitamin replacement.

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Refeeding Risk Criteria (NICE NG22)

A patient is at HIGH RISK if they meet ONE or more of the following:

  • BMI <16 kg/m²
  • Unintentional weight loss >15% in 3–6 months
  • Little or no nutritional intake for >10 days
  • Low levels of potassium, phosphate, or magnesium before refeeding

Patient is at risk if they meet TWO or more of the following:

  • BMI <18.5 kg/m²
  • Unintentional weight loss >10% in 3–6 months
  • Little or no nutritional intake for >5 days
  • History of alcohol misuse or insulin-dependent diabetes, chemotherapy, antacid or diuretic use
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In eating disorders, BMI <16 alone qualifies as high-risk. Do not wait for additional criteria.

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Prevention Protocol — BEFORE Refeeding

  1. Thiamine (Vitamin B1) BEFORE starting nutrition — 200–300 mg oral/day (Pabrinex IV pairs 1+2 twice daily if malabsorption suspected or IV feeding). Must precede any glucose/carbohydrate load to prevent Wernicke's encephalopathy.
  2. Correct potassium to normal range before starting feeding. IV if <3.0 mmol/L.
  3. Correct phosphate — replace with oral phosphate supplement (Phosphate-Sandoz) if <0.8 mmol/L; IV if <0.5 mmol/L.
  4. Correct magnesium — oral magnesium glycerophosphate or IV if symptomatic/severe (<0.5 mmol/L).
  5. Start 5–10 kcal/kg/day (NICE NG22) — not more than 10 kcal/kg/day in very high risk patients. Use 5 kcal/kg if BMI <14 or prolonged starvation.

Thiamine Dose Reminder: Oral thiamine 200–300 mg/day OR Pabrinex IV (Pairs 1+2) twice daily for first 3–5 days if patient severely malnourished or IV nutrition planned. Must be given BEFORE any carbohydrate load.

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Caloric Escalation Protocol

Time Period Target Calories Monitoring Action
Days 1–25–10 kcal/kg/day (maximum)Daily U&E, phosphate, Mg, glucose, ECGThiamine + electrolytes replaced BEFORE starting
Days 3–4Increase by 5 kcal/kg if electrolytes stableContinue daily bloodsVigilant for oedema, tachycardia, respiratory changes
Days 5–7Titrate towards 20 kcal/kg/dayDaily bloods until stableInvolve dietitian — oral diet preferred; NG if unable
Week 2+Advance toward full requirements (25–35 kcal/kg/day)3× weekly bloodsContinue vitamin supplementation; weight monitoring

Phosphate Replacement Threshold: Phosphate <0.6 mmol/L = oral supplementation. Phosphate <0.5 mmol/L = IV replacement required. Replace before and during refeeding. Check phosphate daily for minimum 2 weeks.

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Signs of Refeeding Syndrome

Cardiovascular
  • Cardiac arrhythmias — AF, VT/VF from hypophosphataemia-driven ATP depletion
  • Cardiac failure — rapid fluid shift causes sudden volume overload
  • Sudden unexpected death in first 2 weeks of refeeding
Neurological & Muscular
  • Confusion, delirium — thiamine deficiency, electrolyte disturbance
  • Seizures — hypophosphataemia, hypomagnesaemia
  • Muscle weakness — generalised; respiratory muscle weakness can cause ventilatory failure
  • Wernicke's encephalopathy — ophthalmoplegia, ataxia, confusion (thiamine deficiency)
Fluid & Respiratory
  • Peripheral oedema — sodium and water retention post-refeeding
  • Pulmonary oedema — rapid fluid shift causes respiratory distress
  • Respiratory failure — diaphragm weakness from hypophosphataemia
  • Rising weight from fluid (not fat) in first days — do not increase calories rapidly
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Refeeding Syndrome Risk Screener

Assess Refeeding Risk (NICE NG22 Framework)

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Therapeutic Relationship

Ego-Syntonic Nature of AN

Unlike many psychiatric conditions, AN is often ego-syntonic — the illness aligns with the patient's sense of self and values. Patients typically do not identify the disorder as a problem or desire recovery. This profoundly challenges engagement.

  • Avoid confrontation about weight or eating behaviour
  • Express genuine curiosity about the patient's experience
  • Separate the person from the illness — "the eating disorder" not "you"
  • Acknowledge ambivalence without reinforcing it
Core Nursing Principles
  • Non-judgemental stance — never express disgust, frustration, or moral judgement about eating or weight
  • Empathic validation — acknowledge distress without colluding with eating disorder cognitions
  • Consistency — clear, consistent boundaries around meal plans and expectations
  • Avoid commenting on appearance or weight ("you look better") — reframe around function and wellbeing
  • Document all behaviours objectively without judgement
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Meal Support & Observation

Meal Support Nursing
  • Sit with patient during all meals — supportive presence, not surveillance
  • Maintain calm, neutral demeanour — avoid anxiety about food which the patient will detect
  • Do not comment on what the patient is eating or how much
  • Encourage slow, mindful eating — model calm relationship with food
  • Avoid engaging in debate about calories, weight, or food choices during meals
  • No coercion — physical restraint to force eating is never acceptable outside of legally sanctioned NG feeding
Post-Meal Observation
  • 30–60 minutes post-meal observation — sit with patient to prevent purging
  • Toilet access supervised or delayed post-meal (follow ward protocol)
  • Observe for concealing food (pockets, clothing, disposal)
  • Water loading prior to weigh-ins — observe before weighing if suspected
  • Document observations accurately and non-punitively
  • Observation level (continuous, 15 min, 30 min) documented and reviewed by MDT
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Nasogastric Feeding & Legal Framework

⚖️

NG feeding in the context of meal refusal requires a formal legal and ethical framework. The approach differs between countries but must be documented, proportionate, and regularly reviewed.

Capacity Assessment
  • Assess mental capacity to refuse NG feeding — use Mental Capacity Act (UK) principles or equivalent in GCC jurisdiction
  • AN does not automatically = lack of capacity, but the distorted beliefs may impair decision-making re: nutritional treatment
  • Best interests decision made if lacking capacity — MDT + family + advocacy
  • Capacity assessments must be documented and reviewed regularly
Mental Health Act / Compulsory Treatment
  • Detention under Mental Health Act may enable treatment of AN as the medical consequence of the mental disorder
  • Court orders may be required in some jurisdictions for NG feeding
  • GCC: involuntary psychiatric treatment laws vary — Saudi Arabia, UAE, Qatar, Kuwait have mental health legislation; consult legal/ethics team
  • NG feeding is a nursing clinical skill — requires signed consent or legal authorisation, two-nurse check, correct tube placement confirmation (pH <5.5 or X-ray)
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Weight Restoration & Motivational Interviewing

Weight Restoration Targets
  • Inpatient target: 0.5–1 kg/week
  • Outpatient target: 0.5 kg/week minimum
  • Slower restoration increases refeeding risk paradoxically if too slow to replace depleted phosphate
  • Short-term rapid weight gain in first 1–2 weeks = fluid (not fat) — explain to patient
  • Weekly weigh-in — same time, same conditions, nurse present
Motivational Interviewing (MI)
  • Evidence-based technique for ambivalent patients
  • OARS: Open questions, Affirmations, Reflections, Summaries
  • Explore discrepancy between patient's values and illness impact
  • Roll with resistance — don't argue; reflect ambivalence back
  • Develop change talk — elicit patient's own reasons for recovery
  • Avoid righting reflex (telling patient what to do)
Family-Based Therapy (Maudsley Approach)

FBT is the most evidence-based treatment for adolescent AN. Parents are externalised as agents of recovery, not causes of illness. Three phases: Phase 1 — parents take control of refeeding; Phase 2 — gradual return of control to adolescent; Phase 3 — establishing healthy identity. Nurse's role: support family, psychoeducation, reinforce consistency.

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Safety & Comorbidity

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Eating disorders carry significant suicide risk. AN has a 2–5% suicide mortality. BN and BED carry elevated self-harm risk. All patients require regular risk assessment.

Comorbid Conditions
Depression (50–75% AN) Anxiety disorders (OCD) PTSD / trauma history Autism spectrum Personality disorders Substance misuse (BN)
Risk Assessment Framework
  • Regular safety assessments — minimum weekly inpatient
  • Assess suicidal ideation, self-harm methods, intent, plan
  • Environmental safety — safe area search if high risk
  • Document every assessment, even if low risk — deterioration can be rapid
  • Liaison psychiatry and psychology referral essential
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Multidisciplinary Team (MDT)

Psychiatrist

Leads diagnostic assessment, prescribing (antidepressants, antipsychotics cautiously), MHA/MCA decisions, compulsory treatment, mental health comorbidity management.

ED Nurse Specialist

Coordinates meal support, observation, therapeutic relationship, nursing care plan, patient advocacy, staff education, discharge planning. Key point of contact.

Dietitian

Nutritional assessment, meal planning, refeeding protocol design, caloric targets, NG feed prescribing, vitamin/mineral supplementation, weight monitoring.

Psychologist

CBT-E (Enhanced CBT), FBT facilitation, schema therapy, trauma-focused work, neuropsychological assessment, group therapy, family work.

Physician / Internist

Medical monitoring, electrolyte management, ECG interpretation, cardiac assessment, bone health (DEXA), refeeding syndrome management.

Physiotherapist

Safe graded activity and exercise prescription (avoids compulsive exercise trigger), muscle rehabilitation, bone health programme, body awareness therapy.

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Care Setting Decision Criteria

Setting Criteria for Key Features
OutpatientBMI >15, medically stable, motivated, strong supportWeekly EDNS appointments, dietitian, psychology. GP monitoring. Suitable for BN, BED, mild AN.
Day ProgrammeBMI 13–15, partial engagement, step-up or step-downStructured meal support daytime, therapy groups, overnight at home. 5 days/week intensity.
Inpatient (Psych)BMI <13, MARSIPAN medium-high, treatment refusal, suicide risk24-hour observation, medical monitoring, structured meals, psychological programme.
Inpatient (Medical)Any MARSIPAN HIGH criteria, refeeding, electrolyte instabilityHDU/general medical ward. Stabilise medically before transfer to psychiatric setting.
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Ramadan & Eating Disorders

Ramadan fasting represents a significant clinical risk for patients with eating disorders. Fasting behaviour during Ramadan can mirror and reinforce ED restricting patterns, increasing relapse risk.

  • AN/BN patients should have Ramadan discussed proactively by the MDT — typically 4–6 weeks before Ramadan
  • Many Islamic scholars permit exemption from fasting for medically ill patients (incl. psychiatric disorders) — clinicians should be able to advise on this
  • Risk of using Ramadan as socially sanctioned restricting behaviour — particularly in restrictive AN
  • BN patients: disruption of meal pattern may trigger binge-purge cycles during Iftar
  • Social pressure to attend large Iftar gatherings — anxiety around communal eating
  • Electrolyte monitoring should be maintained during Ramadan in any AN/BN patient who fasts
  • Document Ramadan management plan in care notes; involve family where appropriate
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Male Eating Disorders in GCC

Male eating disorders are significantly underdiagnosed globally and particularly in GCC due to cultural attitudes to masculinity, reduced help-seeking, and clinician bias toward female patients.

Muscle Dysmorphia

"Reverse anorexia" — pathological preoccupation with body being insufficiently muscular. Compulsive exercise, restrictive eating, protein supplement overuse, and anabolic steroid use. High prevalence in GCC gym culture.

Steroid Use

Anabolic steroid use in GCC is increasing among young men seeking muscular physiques. Associated with muscle dysmorphia, cardiac complications (LVH, arrhythmias), hepatotoxicity, psychiatric comorbidity (aggression, depression on cessation).

  • Screen male patients for body image concerns, exercise compulsion, supplement overuse
  • Avoid assuming "he doesn't have an eating disorder" based on gender
  • BMI criteria for AN may not capture musculocentric EDs — use clinical judgement
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Prognosis & Service Context

AN Prognosis
  • 30–40% full recovery
  • 30–40% partial recovery / chronic course
  • 10% mortality (highest of any psychiatric disorder) — cardiac and suicide
  • Longer duration of illness = worse prognosis
  • Adolescent onset with FBT = better prognosis
  • Adult chronic AN — harm reduction model may be appropriate
GCC Service Landscape
  • Specialist eating disorder services are scarce across GCC
  • Most cases managed in general psychiatry or general medical wards without specialist input
  • Saudi Arabia: limited ED-specific inpatient units; referrals often to UK/US for complex cases
  • UAE: some private hospitals (e.g. Priory at Latifa Hospital) offer ED programmes
  • Qatar: HMC developing mental health services — eating disorders emerging priority
  • Strong argument for regional specialist hub-and-spoke model
ℹ️

Transition from adolescent to adult services is a period of high risk. Ensure structured transition planning begins at 16, with named adult clinician from age 17. Avoid any gap in care.

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High-yield exam content for DHA, DOH, SCFHS, QCHP, and MOH nursing licensing exams. Eating disorders appear in psychiatry and medical nursing sections. Focus on diagnostic criteria, refeeding protocol, and electrolyte patterns.

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Diagnostic Criteria — Exam Format

  1. Restriction of energy intake leading to significantly low body weight (BMI <17.5 in ICD-10; DSM-5 does not specify cutoff)
  2. Intense fear of gaining weight or becoming fat, or persistent behaviour interfering with weight gain
  3. Disturbance in the way body weight/shape is experienced; undue influence of weight/shape on self-evaluation; lack of recognition of seriousness of current low body weight
  1. Recurrent binge eating — eating in a discrete time period an amount definitely larger than most people would eat, with sense of lack of control
  2. Recurrent inappropriate compensatory behaviour to prevent weight gain (purging, fasting, excessive exercise)
  3. Binge-purge episodes occur at least once weekly for 3 months
  4. Self-evaluation unduly influenced by body shape and weight
  5. Not occurring exclusively during episodes of AN

ARFID differs from AN: avoidance is driven by sensory features, fear of adverse consequences (choking, vomiting), or lack of interest — NOT by fear of weight gain or distorted body image. Normal BMI possible. Common in children and ASD.

Electrolyte Patterns — High Yield

Electrolyte AN (Restrictive) BN / Purging Refeeding Syndrome
PhosphateLow (starvation)Normal or lowSeverely LOW — hallmark
PotassiumLow-normalSignificantly LOWLow (intracellular shift)
MagnesiumLowLow (laxatives)Low (intracellular shift)
SodiumLow (water loading)Low or normalMay drop (fluid shifts)
GlucoseLow (hypoglycaemia)VariableRises then drops — glucose drive
Acid-baseNormal or acidosisMetabolic ALKALOSISVariable
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Physical Admission Criteria Quick Reference

Any ONE of the following warrants immediate medical admission (MARSIPAN HIGH risk):

BMI<13 kg/m² (adults)
Heart Rate<50 bpm at rest
Systolic BP<90 mmHg
Postural drop>10 mmHg systolic on standing
Temperature<34.5°C
QTc>470 ms on ECG
Muscle functionCannot rise from squat unaided
Rapid weight loss>1 kg/week sustained
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Refeeding Protocol Quick Reference

Step 1 — Before Starting Nutrition
  1. Thiamine 200–300 mg oral/day OR Pabrinex IV (1+2) twice daily
  2. Correct K+, PO4, Mg to normal range (IV if severe)
  3. ECG baseline — note QTc
  4. Blood glucose baseline
Step 2 — Caloric Escalation
  1. Start: 5–10 kcal/kg/day (max 10)
  2. Increase slowly over 4–7 days toward 20 kcal/kg/day
  3. Daily U&E, PO4, Mg, glucose — minimum 2 weeks
  • Target weight gain: 0.5–1 kg/week
  • Mnemonic — REFEEDING: Replace electrolytes first, Escalate calories slowly, Feed orally if possible, ECG and electrolytes daily, Dietitian involvement essential, Involve MDT, NG if necessary, Give thiamine BEFORE glucose load.

    🎯

    GCC Licensing Exam — High-Yield Questions

    Answer: Refeeding syndrome — caused by hypophosphataemia, hypokalaemia, and hypomagnesaemia due to intracellular shift on reintroduction of carbohydrates. Presents with cardiac arrhythmias, respiratory failure, oedema, and Wernicke's encephalopathy. Prevented by slow caloric escalation, thiamine supplementation, and electrolyte correction BEFORE refeeding begins.

    Answer: Phosphate (hypophosphataemia) — phosphate is required for ATP production. Refeeding triggers insulin-mediated intracellular uptake of phosphate, causing plasma levels to fall precipitously. Clinical result: ATP depletion affecting cardiac muscle, diaphragm, and erythrocyte function.

    Answer: Bulimia Nervosa (and AN binge-purge type). Russell's sign refers to calluses or scarring on the dorsal surface of the knuckles (metacarpophalangeal joints) caused by repeated self-induced vomiting. The teeth abrade the skin as fingers are used to stimulate the gag reflex.

    Answer: 0.5–1 kg per week inpatient. Outpatient target is 0.5 kg/week. Rapid weight gain (>1 kg/week) risks refeeding syndrome and is not therapeutic. Too slow (<0.5 kg/week) suggests insufficient nutrition or ongoing compensatory behaviours.

    Answer: Thiamine (Vitamin B1) — thiamine is a co-factor for glucose metabolism (pyruvate dehydrogenase). In thiamine-depleted malnourished patients, administering glucose without thiamine causes accumulation of pyruvate and lactate, leading to Wernicke's encephalopathy (confusion, ophthalmoplegia, ataxia). Dose: 200–300 mg oral/day or Pabrinex IV in severe cases.

    Answer: Immediate medical admission. All three parameters meet MARSIPAN HIGH risk criteria: HR <50, SBP <90, temp <34.5°C. This patient requires urgent medical stabilisation on a general medical ward or HDU — NOT management in the community or psychiatric unit without concurrent medical input. Call senior clinician immediately.