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GCC Nurse — Advanced Clinical Guide

Diabetic Foot — Advanced Nursing Guide

DHA · DOH · SCFHS Exam Ready · MDT-Based Practice
NICE 2023 IDSA CBAHI
Peripheral Neuropathy
The most common component — present in ~50% of diabetes patients over 10 years.
SENSORIMOTOR
  • • Loss of protective sensation (LOPS)
  • • Proprioception impairment
  • • Intrinsic muscle wasting → claw toes
  • • High-pressure areas on plantar surface
  • • Painless — patient unaware of injury
AUTONOMIC
  • • Anhidrosis → dry, cracked skin
  • • AV shunting → warm foot, bounding pulses
  • • Loss of vasomotor tone
  • • Risk of Charcot neuroarthropathy
NEUROPATHIC ULCER FEATURES
  • • Plantar or pressure point location
  • • Painless (patient may not notice)
  • • Callus rim surrounding wound
  • • Well-perfused foot (warm, pulses present)
  • • Punched-out appearance
Peripheral Artery Disease
Accelerated atherosclerosis in diabetes — affects tibial and peroneal vessels predominantly.
ISCHAEMIC ULCER FEATURES
  • • Distal location — tips of toes, heels
  • • Painful (if sensation preserved)
  • • Necrotic base — slough/eschar
  • • No callus formation
  • • Absent or diminished pulses
  • • Cool, pale/mottled foot
  • • Prolonged capillary refill >3 sec
CLINICAL SIGNS OF PAD
  • • Absent DP or PT pulses
  • • Intermittent claudication
  • • Rest pain (critical limb ischaemia)
  • • ABPI <0.9 indicates PAD
  • • ABPI >1.3 = calcified (falsely elevated)
  • • Toe pressures more reliable in DM
MIXED ISCHAEMIC-NEUROPATHIC
Most common presentation in clinical practice. Both components present simultaneously — complex management requiring vascular assessment before aggressive debridement.
Diabetic Foot Infection
Impaired neutrophil function, reduced perfusion, and hyperglycaemia create susceptibility to polymicrobial infection.
MICROBIOLOGY
  • • Polymicrobial — multiple organisms
  • • Gram-positive: S. aureus (dominant)
  • • MRSA risk: previous hospitalisation, prior antibiotics, chronic wounds
  • • Gram-negative: Pseudomonas, coliforms in warm/wet wounds
  • • Anaerobes: deep/necrotic tissue
RED FLAGS
  • • Spreading cellulitis >2cm from wound
  • • Lymphangitis (red tracking lines)
  • • Systemic sepsis: fever, tachycardia, WBC↑
  • • Crepitus (gas-forming organisms)
  • • Deep tracking sinuses
CHARCOT FOOT RECOGNITION
  • • Hot, swollen, red foot in diabetic
  • • May be painless despite severity
  • • X-ray: bony fragmentation/destruction
  • • Do NOT delay offloading — irreversible
  • • MRI preferred for early diagnosis
Neurological Testing Techniques
10g Semmes-Weinstein Monofilament
Apply monofilament perpendicular to skin, hold 1 second — patient identifies site without visual cues.
10 TESTING SITES PER FOOT:
• Plantar hallux• Plantar 2nd toe • 1st met head• 3rd met head • 5th met head• Medial midfoot • Lateral midfoot• Medial heel • Lateral heel• Dorsal 1st web space
Failure at ANY 1 site = peripheral neuropathy present
128 Hz Tuning Fork — Vibration Perception
Strike tuning fork, apply to bony prominence — patient identifies onset and cessation of vibration.
APPLICATION SITES:
  • • Dorsum of distal hallux (primary)
  • • Medial malleolus (if hallux absent)
  • • Tibial tubercle (proximal if needed)
Compare patient's duration of vibration perception with examiner's. Reduced threshold = small fibre neuropathy. Use Biothesiometer for quantitative vibration perception threshold (VPT) — VPT >25V = high ulcer risk.
The Diabetic Foot Triad — Interaction Model
Neuropathy
Undetected trauma
Abnormal pressure
Charcot
Ischaemia
Impaired healing
Tissue necrosis
Gangrene risk
Infection
Polymicrobial
Rapid spread
Osteomyelitis
Each component potentiates the others. Neuropathy allows undetected injury → ulcer formation. Ischaemia impairs healing and immune response. Infection exploits both to spread rapidly through avascular planes. All three must be assessed and managed simultaneously.
GCC Nurse Clinical Guides · Diabetic Foot Advanced Module · Based on NICE NG19, IDSA 2012, IWGDF 2023 guidelines · For educational purposes — always apply local protocols and clinical judgement